Fig. 3From: Targeting mitochondrial biogenesis: a potential approach for preventing and controlling diabetesMitochondrial dysfunction disrupts insulin secretion from pancreatic β-cells. AT 1: Angiotensin II receptor type 1; ROS: Reactive oxygen species; JNK:c-Jun N-terminal kinase; IKK-β: Inhibition of nuclear factor kB kinase; PKC: Protein kinase C; DAG: Diacylglycerol; IRS: Insulin receptor substrateBack to article page